Research
Mortality attributable to nosocomial Clostridium difficile–associated disease during an epidemic caused by a hypervirulent strain in Quebec
Jacques Pépin, Louis Valiquette and Benoit Cossette
CMAJ October 25, 2005 173 (9) 1037-1042; DOI: https://doi.org/10.1503/cmaj.050978
Jacques Pépin
Louis Valiquette
![Loading Loading](https://www.cmaj.ca/sites/all/modules/contrib/panels_ajax_tab/images/loading.gif)
Article Figures & Tables
Figures
Fig. 1: Kaplan–Meier plot showing probability of death since diagnosis among inpatients in whom nosocomial Clostridium difficile–associated disease (CDAD) developed and among matched control subjects without CDAD. No. of days = time since diagnosis of CDAD (cases) or time since reaching the same interval after admission (controls).
Tables
In this issue
Article tools
Respond to this article
Mortality attributable to nosocomial Clostridium difficile–associated disease during an epidemic caused by a hypervirulent strain in Quebec
Jacques Pépin, Louis Valiquette, Benoit Cossette
CMAJ Oct 2005, 173 (9) 1037-1042; DOI: 10.1503/cmaj.050978
Jump to section
Related Articles
Cited By...
- Lysozyme Resistance in Clostridioides difficile Is Dependent on Two Peptidoglycan Deacetylases
- Lysozyme resistance in C. difficile is dependent on two peptidoglycan deacetylases
- A small-molecule antivirulence agent for treating Clostridium difficile infection
- Risk Factors for Acquisition and Loss of Clostridium difficile Colonization in Hospitalized Patients
- Clostridium difficile PCR Ribotype 018, a Successful Epidemic Genotype
- Decreasing Clostridium difficile Infections by an Antimicrobial Stewardship Program That Reduces Moxifloxacin Use
- Epidemic Clostridium difficile Strains Demonstrate Increased Competitive Fitness Compared to Nonepidemic Isolates
- Role of Leptin-Mediated Colonic Inflammation in Defense against Clostridium difficile Colitis
- Variations in Virulence and Molecular Biology among Emerging Strains of Clostridium difficile
- Nontoxigenic Clostridium difficile Protects Hamsters against Challenge with Historic and Epidemic Strains of Toxigenic BI/NAP1/027 C. difficile
- Characterizations of Clinical Isolates of Clostridium difficile by Toxin Genotypes and by Susceptibility to 12 Antimicrobial Agents, Including Fidaxomicin (OPT-80) and Rifaximin: a Multicenter Study in Taiwan
- Clinical and Laboratory Characteristics of Clostridium difficile Infection in Patients with Discordant Diagnostic Test Results
- The effect of hospital-acquired infection with Clostridium difficile on length of stay in hospital
- Lack of Association between Clinical Outcome of Clostridium difficile Infections, Strain Type, and Virulence-Associated Phenotypes
- Prophage-Stimulated Toxin Production in Clostridium difficile NAP1/027 Lysogens
- Neutralization of Clostridium difficile Toxin A with Single-domain Antibodies Targeting the Cell Receptor Binding Domain
- The Changing Epidemiology of Clostridium difficile Infections
- Efficacy of cleaning products for C difficile: Environmental strategies to reduce the spread of Clostridium difficile-associated diarrhea in geriatric rehabilitation
- Multilocus Sequence Typing of Clostridium difficile
- Antiapoptotic Proteins Bcl-2 and Bcl-XL Inhibit Clostridium difficile Toxin A-Induced Cell Death in Human Epithelial Cells
- Effects of Subinhibitory Concentrations of Antibiotics on Colonization Factor Expression by Moxifloxacin-Susceptible and Moxifloxacin-Resistant Clostridium difficile Strains
- Motility and Flagellar Glycosylation in Clostridium difficile
- Antimicrobial resistance in hospitals: How concerned should we be?
- Effects of Exposure of Clostridium difficile PCR Ribotypes 027 and 001 to Fluoroquinolones in a Human Gut Model
- Fairness of financial penalties to improve control of Clostridium difficile
- Clostridium difficile Infections in a Canadian Tertiary Care Hospital before and during a Regional Epidemic Associated with the BI/NAP1/027 Strain
- Rifampin and Rifaximin Resistance in Clinical Isolates of Clostridium difficile
- Tolevamer, an Anionic Polymer, Neutralizes Toxins Produced by the BI/027 Strains of Clostridium difficile
- Increased Sporulation Rate of Epidemic Clostridium difficile Type 027/NAP1
- Use of Highly Discriminatory Fingerprinting to Analyze Clusters of Clostridium difficile Infection Cases Due to Epidemic Ribotype 027 Strains
- The Changing Spectrum of Clostridium Difficile Associated Disease: Implications for Dentistry
- Yield of Stool Culture with Isolate Toxin Testing versus a Two-Step Algorithm Including Stool Toxin Testing for Detection of Toxigenic Clostridium difficile
- Auto-catalytic Cleavage of Clostridium difficile Toxins A and B Depends on Cysteine Protease Activity
- Diarrhoea associated with antibiotic use
- tcdC Genotypes Associated with Severe TcdC Truncation in an Epidemic Clone and Other Strains of Clostridium difficile
- Molecular Analysis of Clostridium difficile PCR Ribotype 027 Isolates from Eastern and Western Canada
- Measuring the presence of chronic diseases
- How should we respond to the highly toxogenic NAP1/ribotype 027 strain of Clostridium difficile?